Fibroblast Growth Factor-23 and Carotid Artery Intima Media Thickness in Chronic Kidney Disease
Özet
Background: The cause of early-accelerated atherosclerosis development observed in Chronic Kidney Disease (CKD) is not fully understood. The determination of the relationship between the levels of fibroblast growth factor 23 (FGF-23) and the development of endothelial dysfunction, left ventricular hypertrophy, and myocardial infarction lends support to the possibility that FGF-23 plays a role in the development of atherosclerosis in CKD. Only a few studies, however, have been conducted that analyze the relationship between FGF-23 levels in the progression of CKD and the development of atherosclerosis, and these studies have generally been limited to those patients receiving dialysis therapy due to end stage renal disease (ESRD).
Anahtar kelimeler
Atıf için künye
Yilmaz, G., Ustundag, S., Temizoz, O., Sut, N., Demir, M., Ermis, V., Sevinç, C., & Ustundag, A. (2015). Fibroblast Growth Factor-23 and Carotid Artery Intima Media Thickness in Chronic Kidney Disease. Clinical laboratory, 61(8), 1061–1070. https://doi.org/10.7754/clin.lab.2015.141236
BibTeX
@article{yilmaz2015-243684,
author = {Yilmaz, Gulay and Ustundag, Sedat and Temizoz, Osman and Sut, Necdet and Demir, Muzaffer and Ermis, Veli and Sevinç, Can and Ustundag, Ayten},
title = {Fibroblast Growth Factor-23 and Carotid Artery Intima Media Thickness in Chronic Kidney Disease},
journal = {Clinical laboratory},
year = {2015},
volume = {61},
number = {8},
pages = {1061--1070},
issn = {1433-6510},
doi = {10.7754/clin.lab.2015.141236},
url = {https://akademik.atauni.edu.tr/yayinlar/243684-fibroblast-growth-factor-23-and-carotid-artery-intima-media-thickness-in-chronic}
}