Makale
Tam Makale
Uluslararası
2026
Hepassocin drives EGFR-dependent endothelial atherogenic activation via NOX1/ROS amplification and p38 signaling.
Archives of Biochemistry and Biophysics
Cilt 781 · 10.04.2026
Atıf için künye
Pyo, M. K., Park, H. S., Ko, J. H., Lim, D. S., Gwon, H. J., Hassıbelnaby, A. M. A., Yağan, R., Song, J., Shin, Y. K., Jeong, J. H., & Jung, T. W. (2026). Hepassocin drives EGFR-dependent endothelial atherogenic activation via NOX1/ROS amplification and p38 signaling. Archives of Biochemistry and Biophysics, 781. https://doi.org/10.1016/j.abb.2026.110820
BibTeX
@article{pyo2026-209934,
author = {Pyo, Min Kyung and Park, Hyung Sub and Ko, Jun Hwi and Lim, Do Su and Gwon, Hyeon Ji and Hassıbelnaby, Abdelaty Mostafa Abdelaty and Yağan, Ridvan and Song, Jin-Ho and Shin, Yong Kyoo and Jeong, Ji Hoon and Jung, Tae Woo},
title = {Hepassocin drives EGFR-dependent endothelial atherogenic activation via NOX1/ROS amplification and p38 signaling.},
journal = {Archives of Biochemistry and Biophysics},
year = {2026},
volume = {781},
issn = {0003-9861},
doi = {10.1016/j.abb.2026.110820},
url = {https://akademik.atauni.edu.tr/yayinlar/209934-hepassocin-drives-egfr-dependent-endothelial-atherogenic-activation-via-nox1ros}
}